在LPS诱导的BV2小胶质细胞炎症模型中，化合物1展现出显著的NO抑制能力（IC 50 =12.0μM），这一发现为开发神经炎症特异性抑制剂提供了分子基础。相关成果发表于《Journal of Asian Natural Products Research》。

hbme 对 lps 诱导的 bv2 小胶质细胞活力的影响：实验结果显示，与对照组相比，lps 处理会降低 bv2 小胶质细胞的活力，而 hbme 处理能够在很大程度上恢复细胞活力，这表明 hbme 对 lps 诱导的小胶质细胞损伤具有保护作用。

离体细胞实验发现脂多糖（lps）显著诱导bv2细胞诱导型一氧化氮合成酶(inos)、环氧合酶-2 (cox-2)和白细胞介素-6（il-6）的表达，在给与cma激活剂后可 ...

In the present study we investigate whether the effects of curcumin on the production of pro-inflammatory mediators in lipopolysaccharide (LPS)-stimulated BV2 microglia.

Schematic representation of the protective mechanisms of resveratrol against the proinflammatory agent mCRP and LPS. The BV2 cell line used herein was established from C57BL/6 transgenic mice ...

To find out, first author Miguel Burguillos and colleagues transfected BV2 microglia cells with small interfering RNAs to knock down endogenous apoptotic caspase-3 or -7. The scientists then ...

BV2 express nuclear v-myc and the cytoplasmic v-raf oncogene products as well as the env gp70 antigen at the surface level. ... (LPS) treatment. Why Choose BV-2 from AcceGen? AcceGen BV-2 cells are ...

Burguillos reasoned that if galectin-3 mediates inflammation, then blocking the protein should dampen microglial responses to LPS. In BV2 cultures, neutralizing galectin-3 with either interfering RNA ...